As nobody else seems to want to help you out here …
Check out activation and mobilis/zation with respect to osteoclasts and osteoclast progenitors, e.g.
“Osteoclast differentiation and activation”http://www.nature.com/nature/journal/v423/n69...
“However, RANKL does not induce OCP mobilization in protein tyrosine phosphatase-ε knockout mice with osteoclasts that are defective in terms of bone adhesion and resorption . Thus, RANKL-induced osteoclast activation may regulate progenitor recruitment as part of homeostasis and host defense, linking bone remodeling with regulation of hematopoiesis.”http://ukpmc.ac.uk/articlerender.cgi?artid=1051388
This basic principle of cellular activity at the remodeling site constitutes the Activation-Resorption-Formation (ARF) sequence (Figure 6).http://www.endotext.org/parathyroid/parathyro...
But you don’t seem to have grasped the basics, RANKL stands for RANK ligand so that what you have written (“ … RANK-Ligand is created that is tied receptor-like to RANKL …
”) would mean that it was tied
[sic] to itself!!!
The term used anyway is not tied
– if you read anything about signalling pathways and receptors you will come across receptor binding
” -> “generated” or simply “produced”
I think you’ve also got the second half of the sentence the wrong way round – have another look at it – it’s not “… also leads to new osteoclasts for recruiting and maturation
” but “ … also leads to the recruitment and maturation of new osteoclasts.” (see second link above)